Park, T. J. Et Al. 2022. α-ketoisocaproic acid promotes ER stress through impairment of autophagy, thereby provoking lipid accumulation and insulin resistance in murine preadipocytes. Biochemical and Biophysical Research Communications , vol.603 , 109-115.

Park, T. J., Park, S. Y., Lee, H. J., HASSIBELNABY, A. M. A., Jeong, J. H., & Jung, T. W., (2022). α-ketoisocaproic acid promotes ER stress through impairment of autophagy, thereby provoking lipid accumulation and insulin resistance in murine preadipocytes. Biochemical and Biophysical Research Communications , vol.603, 109-115.

Park, Tae Et Al. "α-ketoisocaproic acid promotes ER stress through impairment of autophagy, thereby provoking lipid accumulation and insulin resistance in murine preadipocytes," Biochemical and Biophysical Research Communications , vol.603, 109-115, 2022

Park, Tae J. Et Al. "α-ketoisocaproic acid promotes ER stress through impairment of autophagy, thereby provoking lipid accumulation and insulin resistance in murine preadipocytes." Biochemical and Biophysical Research Communications , vol.603, pp.109-115, 2022

Park, T. J. Et Al. (2022) . "α-ketoisocaproic acid promotes ER stress through impairment of autophagy, thereby provoking lipid accumulation and insulin resistance in murine preadipocytes." Biochemical and Biophysical Research Communications , vol.603, pp.109-115.

@article{article, author={Tae Jun Park Et Al. }, title={α-ketoisocaproic acid promotes ER stress through impairment of autophagy, thereby provoking lipid accumulation and insulin resistance in murine preadipocytes}, journal={Biochemical and Biophysical Research Communications}, year=2022, pages={109-115} }