Kandemir, O. Et Al. 2025. Docetaxel-induced liver and kidney toxicity in rats can be alleviated by suppressing oxidative stress, endoplasmic reticulum stress, inflammation, apoptosis and autophagy signaling pathways after Silymarin treatment. Food and Chemical Toxicology , vol.196 .

Kandemir, O., KÜÇÜKLER, S., ÇOMAKLI, S., GÜR, C., & İLERİTÜRK, M., (2025). Docetaxel-induced liver and kidney toxicity in rats can be alleviated by suppressing oxidative stress, endoplasmic reticulum stress, inflammation, apoptosis and autophagy signaling pathways after Silymarin treatment. Food and Chemical Toxicology , vol.196.

Kandemir, Ozge Et Al. "Docetaxel-induced liver and kidney toxicity in rats can be alleviated by suppressing oxidative stress, endoplasmic reticulum stress, inflammation, apoptosis and autophagy signaling pathways after Silymarin treatment," Food and Chemical Toxicology , vol.196, 2025

Kandemir, Ozge Et Al. "Docetaxel-induced liver and kidney toxicity in rats can be alleviated by suppressing oxidative stress, endoplasmic reticulum stress, inflammation, apoptosis and autophagy signaling pathways after Silymarin treatment." Food and Chemical Toxicology , vol.196, 2025

Kandemir, O. Et Al. (2025) . "Docetaxel-induced liver and kidney toxicity in rats can be alleviated by suppressing oxidative stress, endoplasmic reticulum stress, inflammation, apoptosis and autophagy signaling pathways after Silymarin treatment." Food and Chemical Toxicology , vol.196.

@article{article, author={Ozge Kandemir Et Al. }, title={Docetaxel-induced liver and kidney toxicity in rats can be alleviated by suppressing oxidative stress, endoplasmic reticulum stress, inflammation, apoptosis and autophagy signaling pathways after Silymarin treatment}, journal={Food and Chemical Toxicology}, year=2025}