Akademik Veri Yönetim Sistemi
AMERICAN JOURNAL OF EMERGENCY MEDICINE, cilt.34, sa.2, 2016 (SCI-Expanded)
Carbon monoxide (CO) is known as a potent poisonous gas. The clinical features of CO poisoning are not specific. Cardiac disorders due to changes in cellular and subcellular levels, cardiomyopathy, angina pectoris, acute myocardial infarction, arrhythmias, heart failure, pulmonary edema, cardiogenic shock, and sudden death may occur. Myocardial damage has been determined as an indicator of increased mortality in patient with CO poisoning in the long term. The electrocardiographic (ECG) changes that may be detected in carbon monoxide intoxication are repolarization disorders, flattening and inversion of T waves, ischemic changes, QT-interval prolongation, P-wave elevation, QRS widening, and new-onset bundle-branch block in 2 consecutive derivations. We aimed to present the development of a transient left bundle-branch block due to CO poisoning. A 48-year-old female patient was admitted to emergency service with complaints of headache, nausea, and somnolence. Carboxyhemoglobin was 14.8%. In ECG, left bundle-branch block was present. Her biochemical laboratory test results revealed troponin I as 0.05 (normal value, <0.04). In ECG obtained during consultation with Cardiology, normal sinus rhythm was identified. The patient whose coronary angiography findings were interpreted as normal and who had no additional complaint during follow-up was discharged from the hospital. Consequently, our case is the first and only case in whom transient left bundle-branch block had developed due to CO poisoning; and this left bundle-branch block was proven to be CO-induced by normal coronary angiography.